Mamta Vijay Behl Graduate Student Phone: (765) 496-3555 E-mail: mbehl@purdue.edu Full directory listing

Education

Research

Research interests include studying the effects of occupational and environmental exposure to metals in neurodegenerative diseases at the brain barriers.

Ph.D Dissertation: "Effect of Lead Exposure on beta - amyloid clearance at the Blood-CSF-Barrier: Implications in Alzheimer's disease"

Service and Engagement

• Purdue Graduate Student Government- Travel Grants and Student Affairs Committees
• Task Force for Technology in Graduate Student Recruiting Committee- Pharmacy, Nursing and Health Sciences
• School of Health Science Awards Committee
• Grade Appeal Hearing Committee- PNHS
• President of Purdue Meditation
• Volunteer- Lafayette Mental Health Center, West Lafayette, Indiana.

Teaching

• HLS 101- Introduction to Health Science Professions
• PSY 333- Motivation
• PSY 120- Introduction to Psychology

Honors and Credentials

• Graduate Student Service Award, School of Health Sciences, Purdue University, Indiana, April 2009.
• Travel Grant awarded by the Society of Toxicology, Baltimore, MD. March 2009.
• Association of Scientists of Indian Origin SIG- SOT Mehendale and Singh Student/ Postdoctoral-Young Investigator Merit Award, Society of Toxicology, Baltimore, MD. March 2009.
• Second place in the Metal Specialty Section Graduate Student award, Society Of Toxicology, Baltimore, MD, March 2009.
• Travel Grant awarded by the American College of Toxicology Tucson, AZ, November 2008.
• Principal K.M.Kundnani Gold medal for All Round performance, Mumbai, India, August 2001.

Presentations

Behl M, Zhang Y & Zheng W. 'Lead Exposure increases beta-amyloid in the blood cerebrospinal fluid barrier (BCB) by altering efflux transporter Lipoprotein Receptor Protein-1 (LRP-1)'. American Society of Toxicology, Tuscon, AZ, November 2008.

Behl M, Zhang Y & Zheng W. 'Acute exposure to lead results in an increased accumulation of beta-amyloid in the choroid plexus by potentially altering transporter low density lipoprotein receptor protein-1(LRP1)'. Society of Toxicology, Baltimore, MD, March, 2009.

Zhang Y, Behl M, Fan Q & Zheng W. 'Lead Exposure Decreases Copper Clearance fromthe cerebrospinal Fluid (CSF) by the Blood-CSF Barrier'. Society of Toxicology, Baltimore, MD, March, 2009.

Monnot A, Behl M, Zhang Y & Zheng W. 'Regulation of ATP7A in Copper Homeostatsis as Affected by Iron Status in the Blood-Cerebrospinal Fluid Barrier. Society of Toxicology, Baltimore, MD, March,2009.

Behl M, Walls EK & Davidson TL. 'Hippocampal protein synthesis and the extinction of appetitive behavior'. Annual Proceedings of Neuroscience Convention, Washington D.C, November 2005.

Behl M. 'Hippocampal protein synthesis and the extinction of appetitive behavior'. 18th annual Tri State Plus conference on Animal Learning, Behavior & Cognition, University of Windsor, Canada, May 2005.

Behl M & Davidson TL. ' A role for the hippocampus in the inhibitory control of food intake and body weight gain'. Annual Proceedings of Neuroscience Convention, San Diego, CA, November 2004.

Chan KH, Davidson TL, Jarrard LE & Behl M. 'Effects of a longer ISI on hippocampal lesioned rats and simple Pavlovian conditioning'. Annual Proceedings of Neuroscience Convention, New Orleans, LA,November 2003.

Representative Publications

Behl M, Zhang Y and Zheng W (2009). Involvement of insulin-degrading enzyme in the clearance of beta-amyloid at the blood-CSF barrier: Consequences of lead exposure. Cerebrospinal Fluid Res. 6:11.

Behl M, Zhang Y, Monnot AD, Jiang W and Zheng W (2009). Increased beta-amyloid levels in the choroid plexus following lead exposure and the involvement of low-density lipoprotein receptor protein-1. Toxicol Appl Pharmacol. 240 (2):245-54.

Behl M, Zhang Y & Zheng W. 'Lead increases retention of beta-amyloid at the choroid plexus by altering the localization of low density lipoprotein receptor protein-1 (LRP1) via Protein Kinase C' . Manuscript in preparation.

Monnot A, Behl M, Zhang Y & Zheng W. 'Regulation of ATP7A in Copper Homeostatsis is altered by Iron Status in the Blood-Cerebrospinal Fluid Barrier'. Manuscript in preparation.